Gastroparesis is a condition in which the stomach motility (spontaneous contraction of the muscles) does not function properly. When stomach muscles are strong and healthy, they push the food forward along the digestive tract, until it is ready to be released through the bowels. In gastroparesis, the motility is limited or does not work at all, so the stomach does not empty completely. This way, gastroparesis interferes with normal digestion, leading to nausea or vomiting.
When the cause of gastroparesis is unknown (in most cases), the condition is referred to as idiopathic gastroparesis. When the condition develops in diabetics, it is known as diabetic gastroparesis.
Patients recovering from surgery can develop gastroparesis as well.
There is currently no cure for gastroparesis, but there are treatment options available to help ease the condition. Treatment is usually a combination of dietary changes and medications.
As mentioned, in most cases, the cause of gastroparesis is unknown. Gastroparesis is believed to be triggered by a problem with the nerves and muscles that control the stomach emptying. Nerve damage and muscle malfunctioning impact the motility, so food doesn’t completely leave the stomach.
Other possible causes of gastroparesis include taking certain painkillers, having Parkinson’s disease, scleroderma, and amyloidosis (a group of rare diseases caused by deposits of abnormal protein in tissues and organs).
Symptoms of gastroparesis include:
Complications resulting from gastroparesis include dehydration from vomiting, gastro-esophageal reflux disease (GERD), malnutrition, unpredictable sugar levels (a serious risk for diabetics), undigested food that remains in the stomach, and a decreased quality of life.
Risk factors for gastroparesis include having diabetes, hypothyroidism, or infection, recovering from abdominal surgery, taking certain medications (especially those that slow down stomach motility), undergoing certain cancer treatments, and having scleroderma or nervous system diseases like Parkinson’s disease and multiple sclerosis.
A combination of several tests is required to properly diagnose gastroparesis, including upper gastrointestinal endoscopy, CT enterography, and MR enterography, X-rays of the gastrointestinal tract, gastric emptying study, and a breath test measuring the amount of gas produced after drinking sugar water and released through the breath.
The primary mode of treatment for gastroparesis involves changing your diet. Diet changes for gastroparesis include eating smaller meals more frequently throughout the day, chewing your food thoroughly, eating vegetables that are well cooked as opposed to raw, avoiding fibrous food like broccoli or oranges, choosing low-fat foods, consuming pureed food or soups, drinking adequate amounts of water with each meal, exercising gently after each meal, and avoiding carbonated beverages, alcohol, and smoking.
Drugs for gastroparesis include medications to stimulate the stomach muscles and medications to control nausea and vomiting.
In some cases, surgery is required to treat gastroparesis if the patient is unable to tolerate food and liquids. Doctors may recommend a feeding tube to be placed in the small intestine, or a gastric venting tube to help relieve pressure from gastric contents.
Feeding tubes are temporary and are fed through the nose into the small intestine. They are used in severe cases of gastroparesis.
Your doctor will determine the best mode of treatment depending on the severity of your condition.